quinta-feira, 22 de dezembro de 2005

Cuidado com as vacinas!

Esta praticamente obrigatoriedade de vacinarmos as nossas crianças, apesar das leis portuguesas não nos obrigarem a tal, faz muito mal às nossas crianças.
É possível não vacinar os nossos filhos.
Os pais que façam uma declaração escrita para tal, podem assim proteger os seus filhos desta violência.
Se for mesmo perseguido por quem se arroga com direitos para tal, apesar de não os ter, há forma de inibir este horror com remédios homeopáticos. Nas escolas privadas costuma ser pior.
Aqui estão alguns links sobre vacinação e seus perigos que acho bastante interessantes.
Para saber mais clique aqui, ou aqui, ou aqui ou ainda aqui.
Contém informação que era bom que mesmo os pais interessados em vacinar os filhos lessem... Muita atenção ao que está incluído no capítulo "Recommendations for safe vaccination".
Triste, triste, é ver as crianças a sofrerem com os efeitos secundários da vacinação, enquanto as pessoas já estarem a achar normal e natural que um bebé tenha os mais variados tipos de reacções adversas à vacinação... e nem sequer se questionem sobre o assunto!
Esperemos que as coisas mudem.
Ou pelo menos que vão mudando... já era bem bom.

8 comentários:

Anónimo disse...

Quaker 100% Natural Oats & Honey Granola
A half cup of these is coated with three teaspoons of sugar and laden with more artery clogging fat than you'd get in a McDonald's hamburger. Better choices are, Grape Nuts, Wheaties, Kellogg's All-Bran, Post 100% Bran,shredded wheat, or oatmeal.

2. Bugles
These are fried in highly saturated coconut oil. This is about twice as saturated as lard. One serving (just over a cup) gives you 40% of your daily limit of saturated fat. Baked Bugles or tortilla chips are a much better choice.

3. Buitoni Contadina Alfredo Sauce
Does a third of a stick of butter sound good for you? That is how much is in this sauce.Try Classico Spicy Red Pepper, Tomato & Basil, Fire Roasted Tomato & Garlic, or any sauces from Healthy Choice or Ragu Light. Your arteries will thank you.

4. Pizza Hut's Big New Yorker Pizza
Is bigger really better? NO! Two slices of this pizza gives you almost a full day's saturated fat (17grams) and sodium(2.200mg), and 790 calories. And that's without sausage, pepperoni, or anything else. An entire Healthy Choice Supreme French Bread Pizza will only get you 1.5 grams of saturated fat, 580 mg of sodium and 330 calories.

5. Entenmann's Rich Frosted Donut (Variety pack size)
Is it possible than such a yummy snack can have as much artery clogging saturated and trans fat (10grams) as nine strips of bacon? It sure is! The Entenmann's Light Donuts are a better choice, with anywhere from six to nine grams of fat per donut. Though certainly not a healthy food, they are better than the regular ones.

6. Nissin Cup Noodles with Shrimp
These are pre-fried in palm oil, and will clog your arteries up the same as one and a half cups of whole milk. Fantastic Foods Chicken Free Ramen Noodles are a better pick in this department.

7. Burger King French Fries
This franchise makes some of the worst french fries you can buy at a fast food restaurant.They are even worse than McDonald's Super Size Fries. The salty coating allows more oil to be absorbed. A king size order of BK fries packs a punch with 590 calories and 30 grams of fat,12 of them artery clogging.

8. Campbell's red and white label condensed soups
Nothing like a hot bowl of soup on a cold day? True, but these have 1,100mg of sodium, about half the ideal quota for one day. Healthy Choice and Campbell's Healthy Request have less than half as much sodium without sacrificing any taste.

9. Frito Lay's Wow! Potato Chips
These are fried in Olean, the indigestible fat substitute. It doesn't provide any calories, but many have suffered such severe cramps or diarrhea that they had to go to the emergency room! It also prevents the body's absorption of carotenoids.Again, try baked potato or tortilla chips.

10. Denny's Grand Slam
2 eggs, 2 sausage links, 2 strips of bacon, and 2 pancakes. Sounds delicious right? Well, just listen to this. It contains three quarters of a day's total fat (50 grams) and saturated fat (14 grams), nearly a full day's sodium (2,240mg) and one and a half day's cholesterol (460mg). In contrast, the Denny's Slim Slam slashes the calories to 600, the fat to 12 grams, the saturated fat to 3 grams, and the cholesterol to a mere 35 mg.

Anónimo disse...

Concordo com o que diz aqui. São até muito piores do que aqui diz.
Veja aqui
"Thiomersal is a preservative containing small amounts of
ethylmercury that is used in routine vaccines for infants and children. The effect of vaccines containing thiomersal on concentrations of mercury in infants¡¯ blood has not been extensively assessed, and the metabolism of ethylmercury in infants is unknown.
We aimed to measure concentrations of mercury in blood, urine, and
stools of infants who received such vaccines.

Methods
40 full-term infants aged 6 months and younger were given vaccines
that contained thiomersal (diptheria-tetanus-acellular pertussis vaccine, hepatitis B vaccine, and in some children Haemophilus influenzae type b vaccine). 21 control infants received thiomersal-free vaccines. We obtained samples of blood, urine, and stools 3¨C28 days after vaccination. Total mercury (organic and inorganic) in the samples was measured by cold vapour atomic absorption.

Findings
Mean mercury doses in infants exposed to thiomersal were 45¡¤6 ¦Ìg
(range 37¡¤5-62¡¤5) for 2-month-olds and 111¡¤3 ¦Ìg (range 87¡¤5-175¡¤0)
for 6-month-olds. Blood mercury in thiomersal-exposed 2-month-olds
ranged from less than 3¡¤75 to 20¡¤55 nmol/L (parts per billion); in 6-month-olds all values were lower than 7¡¤50 nmol/L. Only one of 15 blood samples from controls contained quantifiable mercury.
Concentrations of mercury were low in urine after vaccination but
were high in stools of thiomersal-exposed 2-month-olds (mean 82 ng/g
dry weight) and in 6-month-olds (mean 58 ng/g dry weight). Estimated
blood half-life of ethylmercury was 7 days (95% Cl 4¨C10 days).

Interpretation
Administration of vaccines containing thiomersal does not seem to raise blood concentrations of mercury above safe values in infants.
Ethylmercury seems to be eliminated from blood rapidly via the stools after parenteral administration of thiomersal in vaccines.

Introduction
Thiomersal is a preservative used in vaccines routinely administered
to infants and children. Its antimicrobial activity is due to small amounts of ethylmercury; the usual dose of paediatric vaccine contains 12¡¤5¨C25 ¦Ìg of mercury.1¨C3 When vaccines ontaining thiomersal are administered in the recommended doses, allergic reactions have been rarely noted, but no other harmful effects have been reported.4 Massive overdoses from inappropriate use of products containing thiomersal have resulted in toxic effects.5¨C9

Mercury occurs in three forms: the metallic element, inorganic salts, and organic compounds (eg, methylmercury, ethylmercury, and
phenylmercury). The toxicity of mercury is complex and dependent on
the form of mercury, route of entry, dosage, and age at exposure.
Mercury is present in the nvironment in inorganic and organic forms, and everyone is exposed to small amounts.10,11 The main route of environmental exposure to organic mercury is consumption of predatory fish, especially shark and wordfish. A 6-ounce can of tuna contains 2¨C127 ¦Ìg (average 17 ¦Ìg) of mercury.12 Freshwater fish (eg, walleye, pike, muskie, and bass) can also contain high concentrations of mercury.

Most of the toxic effects of organic mercury compounds take place in
the central nervous system, although the kidneys and immune system
can also be affected.10,11,13 Organic mercury readily crosses the
blood-brain barrier, and fetuses are more sensitive to mercury exposure than are children or adults. Data about potential differences in toxicity between ethylmercury and methylmercury are few. Both are associated with neurotoxicity in high doses; in-utero poisoning with methylmercury causes problems that are similar to cerebral palsy. Findings about the effect of low-dose methylmercury exposure on neurodevelopment in infants are contradictory.14,15 In-
utero exposure could be related to subtle neurodevelopmental effects
(eg, on attention, language, and memory) that can be detected by
sophisticated neuropsychometric tests¨Calthough the conclusion is
confounded by concomitant ingestion of polychlorinated biphenyls in
the patients investigated.14,15

No toxic effects of low-dose exposure to thiomersal in children have been reported.3 The effect of the small amounts of mercury contained in vaccines on concentrations of mercury in infants¡¯ blood has not been extensively assessed, and the metabolism of ethylmercury in infants is unknown. We aimed to assess concentrations of mercury in
full-term infants after administration of routine vaccinations according to the schedule used in the USA, and to obtain additional information about the presence of mercury at other body sites including urine and stool. Samples of hair and breast milk were also obtained from some mothers of infants participating in the study.

Methods
Study populations
We studied two groups of full-term infants who differed in their
history of exposure to vaccines containing thiomersal. Infants in
the exposure group were recruited at the Elmwood Pediatric Group, a
large paediatric practice in Rochester, NY, USA, where vaccinations with thiomersal preservative were routinely given. 20 infants aged 2 months and 20 aged 6 months were studied at this practice to obtain information about the range of total thiomersal exposures likely to take place during infancy. The control group consisted of 21 infants who did not receive vaccines containing thiomersal and were
recruited from the National Naval Medical Center, Bethesda, MD. All
the infants were recruited during routine well-child examination and
vaccination visits by the investigators (between November, 1999 and October, 2000). Written informed consent was obtained from parents for all procedures.

Vaccines
Vaccines containing thiomersal that were given to infants in the
exposure group included Tripedia (diphtheria-tetanus-acellular
pertussis vaccine; Aventis Pasteur, Swiftwater, PA; 0¡¤01% thiomersal, 25 ¦Ìg mercury per dose) Engerix (hepatitis B vaccine;
GlaxoSmithKline, Rixensart, Belgium; 0¡¤005% thiomersal, 12¡¤5 ¦Ìg
mercury per dose), and in some children HibTITER (Haemophilus
influenzae type b conjugate vaccine, Wyeth-Lederle, Pearl River, NY,
USA; 0¡¤01% thiomersal, 25 ¦Ìg mercury per dose). Vaccines
administered to the control group included Infanix (diptheria-
tetanus-acellular pertussis vaccine; GlaxoSmithKline, Rixensart,
Belgium), Recombivax HB (hepatitis B vaccine; Merck, West Point, PA,
USA), and ActHIB (Haemophilus influenzae b conjugate vaccine,
Aventis Pasteur, Swiftwater, PA, USA).
Procedures
We obtained vaccination istories¡ªincluding type of vaccine, manufacturer, lot number, and dates of administration¡ªfrom the medical records. In the exposure group, we obtained samples of
heparinised whole blood, stool, and urine, during a visit 3¨C28 days
after vaccination. Blood and urine were kept at 4¡ãC, and stools were
frozen until assessment. Urine was sampled by use of a urine bag at
the clinic, and stool was taken from a diaper (nappy) provided by
the parent. Whole blood and urine were obtained from the control
children. At both sites, we obtained at least 50 hairs from the mother by cutting at the base near the scalp in the occipital area, to assess potential transplacental exposure of infants to mercury.
Additionally, several samples of breastmilk or formula were obtained
from mothers of infants at Elmwood Pediatric Group, as well as stool
samples from a few infants who were not exposed to thiomersal.
We measured total mercury in all samples (and inorganic mercury in
stool samples) by cold vapour atomic absorption as previously
described.16,17 The limit of reliable quantitation in this assay
ranged between 7¡¤50 nmol/L and 2¡¤50 nmol/L, dependant on sample
volume.
Population pharmacokinetic calculations
To estimate the half-life of thiomersal mercury in the blood, we
developed a prediction model for the expected concentrations of
mercury in blood for half-lives of mercury ranging from 1 day to 45
days, on the basis of bodyweight of the infant, the doses of thiomersal administered, and the times between the individual doses of thiomersal and when the blood was obtained. To do these calculations, we assumed that 5% of the mercury dose was distributed to blood,7 that blood volume represented about 8% of the infant's bodyweight, and that elimination of mercury from blood followed a
single-compartment model with first-order kinetics. For each
possible half-life between 1 and 45 days, we then calculated the
difference between the predicted and actual recorded concentrations
in blood for each infant. Only measurements within the range of
reliable quantitation were used in these calculations.
The best estimate of the blood half-life of mercury was judged to be the hypothetical half-life, which resulted in the smallest difference between predicted and observed values. We constructed a 95% CI based on a likelihood ratio for this estimate with the assumption that errors from the decay model were independent, additive, and normally distributed. The 95% confidence limits were
the points where the curve crossed the minimum sum of squares
multiplied by 1 + ¦Ö2(I)/(n−1) where n is the number of data points
and ¦Ö2(1) is the upper 5% point of the ¦Ö2 distribution on one degree
of freedom.

Statistical analysis
Because this was a descriptive study we did no formal calculations
for sample size. Student's t test and Fisher's exact test were used
to compare results for the exposure and control group, with p¡Ü0¡¤05
judged to be significant.

Role of the funding source
The sponsors of the study approved the study design but had no other
involvement in the in study design, data collection, data analysis,
data interpretation, or writing of the report.

Results
61 infants were enrolled in this study (table). Among infants aged 2
months in the exposure group, samples were taken from eight within 7 days of vaccination, from five between 8 and 14 days after
vaccination, and from seven between 15 and 21 days after vaccination. Among 6-month-old infants in the exposure group,
samples were taken from seven between 4 and 7 days after
vaccination, from eight between 8 and 14 days after vaccination, and
from five between 15 and 27 days after vaccination. Samples were
obtained from infants in the control group at regularly scheduled
visits at 2 or 6 months of age. All children remained healthy
throughout the study and during 24¨C36 months of follow-up.

Table Concentrations of mercury in blood, urine, and stool of
infants who received vaccines containing thiomersal and those who
did not.
Sufficient volumes of blood (¡Ý 1 mL) for the measurement of mercury
by the atomic absorption technique were obtained from 17 infants
aged 2 months and 16 aged 6 months in the exposure group. Mercury
concentrations were below the range of reliable quantitation in five
of 17 blood samples from 2-month-olds, and seven of 16 blood samples from 6-month olds (p=0¡¤48). The mean concentration of blood mercury in samples with quantifiable mercury was higher in 2-month-olds than in 6-month olds (difference 3¡¤05 nmol/L, 95% CI 0¡¤03¨C1¡¤24, p=0¡¤06), but was low in both these groups (table). Sufficient blood volumes
for measurement of mercury were obtained from 15 infants in the
control group, including eight aged 2 months and seven aged 6
months. Blood mercury was below the level of reliable quantitation
in seven of the eight samples from the 2-month-olds and in all seven
samples from 6-month-olds. The only detectable value from the control group was 4¡¤65 nmol/L.
Overall, mercury concentrations were below the range of quantitation
in 12 of 33 samples from thiomersal-exposed infants and in 14 of 15 unexposed infants (p=0¡¤04). The highest level of blood mercury
detected in any infant in this study was 20¡¤55 nmol/L, which was
measured 5 days after vaccination in a 2-month-old infant weighing
5¡¤3 kg, who had received vaccines (Tripedia and Engerix B)
containing a total dose of 37¡¤5 ¦Ìg mercury. The relation between
time between vaccination and sampling and the concentration of
mercury in the blood in the exposed group is shown in figure 1.
Although mercury concentrations were uniformly low, the highest
levels were recorded soon after vaccination.

Figure 1. Blood mercury concentrations in Infants aged 2 months (diamonds) and 6 months (squares) by time of sampling.
Filled symbols represent measured values and open symbols represent
samples at the limit of quantitation, either 7¡¤50 nmol/L, 3¡¤75 nmol/L, or 2¡¤5 nmol/L, dependent on sample volume.
Mercury was undetectable in most of the urine samples from the
infants in this study. Only one of 12 urine samples from 2-month-
olds, and three of 15 from 6-month-olds in the exposure group, and none of the 14 samples from the controls, contained detectable
mercury. The highest concentration of urinary mercury detected was
6¡¤45 nmol/L, in a 6-month old infant in the exposure group (table).
Stool samples were collected from infants in the exposure group. All
of the stool samples from infants who received thiomersal-containing
vaccines had detectable mercury, with concentrations in stools from
2-month-old infants slightly higher than those in 6-month-olds (p=0¡ ¤098, table). As expected, most of the mercury in stools was
inorganic. Stool samples were not obtained from control infants;
therefore, to determine whether dietary intake could contribute to
the mercury content of stools, we also obtained samples from nine
infants at Elmwood Pediatric Group who were age-matched with the
infants in the exposure group and were not exposed to vaccines
containing thiomersal. The mean mercury concentration in the stools
of these infants was 22 ng/g dry weight (SD 16), which was
significantly lower (p=0¡¤002) than the mean of the samples collected
from thiomersal-exposed infants.
Amounts of mercury measured in maternal hair are shown in figure 2.
The mean concentration of hair mercury in mothers of the exposure
group was 0¡¤45 ¦Ìg/g hair, whereas the mean amount in mothers of the
control infants was 0¡¤32 ¦Ìg/g (p=0¡¤22). Eight mothers of infants in the 6-month-old cohort provided breast milk samples. Concentrations
of mercury in these samples were low (mean=0¡¤30 ¦Ìg/g, range 0¡¤24¨C
.

Figure 2. Mercury concentrations in hair from mothers of infants.
Bar represents mean concentration of mercury in maternal hair.
We estimated the half-life of mercury in blood after vaccination to be 7 days, since this result gave the smallest difference between
the expected and recorded (measured) concentration (figure 3). The
95% CI around this estimate was 4¨C10 days. The half-life estimate
was very similar when only measurements in 2-month-olds (7 days, 95%
CI 4¨C11) or 6-month-olds (5 days, 3¨C9) were included, suggesting
that the rate of elimination of thiomersal mercury from blood was
similar in both age-groups.

Figure 3. Estimated blood half-life of mercury in infants who were
exposed to thiomersal.
Lines represent sum of square of differences between observed
concentrations of blood mercury (nmol/L) and those predicted for
every individual infant on the basis of bodyweight and time of sampling, with a series of hypothetical half-lives shown on x axis.
Arrow shows point with lowest value for squared difference,
indicating best estimate for serum half-life.

Discussion
We have shown that very low concentrations of blood mercury can be
detected in infants aged 2¨C6 months who have been given vaccines
containing thiomersal. However, no children had a concentration of
blood mercury exceeding 29 nmol/L (parts per billion), which is the
concentration thought to be safe in cord blood;18 this value was set
at ten times below the lower 95% CI limit of the minimal cord blood
concentration associated with an increase in the prevalence of
abnormal scores on cognitive function tests in children. Blood
mercury concentrations indicate concentrations in organs well.18
Although our study was not designed as a formal assessment of the
pharmacokinetics of mercury, we did obtain samples of blood at
various time points after exposure. Assessment of these samples
suggested that the blood half-life of ethylmercury in infants might
differ from the 40¨C50 day half-life of methylmercury (range 20¨C70
days) in adults and breastfeeding infants.10,19 The concentrations
of blood mercury 2¨C3 weeks after vaccination noted in our study were
not consistent with such a long half-life, but suggested a half-life
of less than 10 days. However, this conclusion is based on several
assumptions and a very simple model, and does not take into account
the fact that at least some of the mercury detected in the blood of
the infants in this study is likely to have been derived from
exposures other than vaccination. Because of the short period
between vaccination and sampling, the findings of Strajich and
colleagues20 could be consistent with either a 6-day or 40-day half-
life, but are otherwise consistent with the assumptions made in our
model. Because we expected a 45-day half-life on the basis of
methylmercury pharmacokinetics, the first blood samples were
obtained 3 days after vaccination. Blood samples taken in the first
72 hours after vaccination, stool samples obtained every 24 h, and
samples from premature newborn babies (weighing ¡Ý2000 g) given a
birth dose of hepatitis B vaccine would have helped us to reach
stronger conclusions. Thus, additional studies of the pharmacology of thiomersal in infants are underway.
At the times tested after vaccination, mercury excretion in urine in our study population was low. By contrast, concentrations of mercury
in stool were high, and combined with the finding that stool mercury
concentrations in infants who were not exposed to thiomersal were
significantly lower is consistent with the hypothesis that the
gastrointestinal tract represents a possible mode of elimination of
thiomersal mercury in infants.
Overall, the results of this study show that amounts of mercury in
the blood of infants receiving vaccines formulated with thiomersal
are well below concentrations potentially associated with toxic
effects. Coupled with 60 years of experience with administration of
thiomersal-containing vaccines, we conclude that the thiomersal in
routine vaccines poses very little risk to full-term infants, but
that thiomersal-containing vaccines should not be administered at
birth to very low birthweight premature infants. Decisions about the elimination of thiomersal from these vaccines must balance the
potential benefit of reduced exposure to mercury against the risks of decreased vaccine coverage because of higher costs, the risk of
sepsis in recipients because of bacterial contamination of
preservative-free formulations, and the risks of exposure to
alternative preservatives that might replace thiomersal.

Conflict of interest statement

None declared.
Contributors

M Pichichero and J Treanor contributed to the study conception and
design; obtained, assessed, and interpreted data; drafted and
revised the manuscript; and provided statistical expertise and
supervision.
E Cernichiari contributed to analysis and interpretation of data,
revision of the manuscript, and technical support. J Lopreiato
contributed to revision of the manuscript, and obtained data.

Acknowledgments

We thank Tom Clarkson for advice about the interpretation of mercury
assays, David Oakes for statistical advice, Doreen Francis for
recruiting participants and obtaining samples, and Margaret Langdon
and Nicole Zur for technical assistance. The investigation was
funded by the US National Institutes of Health (NIH), Bethesda, MD,
under contract 1 AF-45248.

References
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Lancet 2000; 355: 1279-1280. Full Text | PDF (47 KB) | MEDLINE |
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Committee on Environmental Health. Thimerosal in vaccines¡ªAn interim
report to clinicians. Pediatrics 1999; 104: 570-574. MEDLINE |
CrossRef
3. Ball LK, Ball R, Pratt RD. An assessment of thimerosal use in
childhood vaccines. Pediatrics 2001; 107: 1147-1154. CrossRef
4. Cox NH, Forsyth A. Thimerosal allergy and vaccination reactions.
Contact Dermatitis 1988; 18: 229-233. MEDLINE
5. Axton JH. Six cases of poisoning after a parenteral organic
mercurial compound (merthiolate). Postgrad Med J 1972; 561: 417-421.
6. Fagan DG, Pritchard JS, Clarkson TW, Greenwood MR. Organ mercury
levels in infants with omphaloceles treated with organic mercurial
antiseptic. Arch Dis Child 1977; 52: 962-964.
7. Matheson DS, Clarkson TW, Gelfand EW. Mercury toxicity
(acrodynia) induced by long-term injection of gammaglobulin. J
Pediatr 1980; 97: 153-155. MEDLINE
8. Lowell JA, Burgess S, Shenoy S, Curci JA, Peters M, Howard TK.
Mercury poisoning associated with high-dose hepatitis-B immune
globulin administration after liver transplantation for chronic
hepatitis B. Liver Transpl Surg 1996; 2: 475-478. MEDLINE
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Environ Health Perspect 1992; 100: 31-38. MEDLINE
11. Clarkson TW. The toxicology of mercury. Crit Rev Clin Lab Sci
1977; 34: 369-403. MEDLINE
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in canned tuna. J AOAC Int 1993; 76: 36-38. MEDLINE
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causes human T-cells to undergo apoptosis: evidence of mitochondrial
dysfunction. Environ Res 1998; 77: 149-159. MEDLINE | CrossRef
14. Grandjean P, Weihe P, White RF, et al. Cognitive deficit in 7-
year-old children with prenatal exposure to methylmercury.
Neurotoxicol Teratol 1997; 6: 417-428.
15. Davidson PW, Myers GJ, Cox C, et al. Effects of prenatal and
postnatal methylmercury exposure from fish consumption on
neurodevelopment: outcomes at 66 months of age in the Seychelles
child development study. JAMA 1998; 280: 701-707. MEDLINE | CrossRef
16. Cernichiari E, Toribara TY, Liang L, et al. The biological
monitoring of mercury in the Seychelles study. Neurotoxicology 1995;
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17. Cernichiari E, Brewer R, Myers GJ, et al. Monitoring
methylmercury during pregnancy: maternal hair predicts fetal brain
exposure. Neurotoxicology 1995; 16: 705-710. MEDLINE
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19. National Academy of Sciences. Toxicologic effects of
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mercury after hepatitis B vaccination in preterm infants. J Pediatr
2000; 136: 679-681. Abstract | Full Text | MEDLINE
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Affiliations
a Department of Microbiology/Immunology, University of Rochester,
Rochester, New York, NY, USA
b Department of Environmental Medicine, University of Rochester,
Rochester, New York, NY, USA
c Department of Medicine, University of Rochester, Rochester, New
York, NY, USA
d National Naval Medical Center, Bethesda, MD
Correspondence to: Dr Michael E Pichichero, Department of
Microbiology/Immunology, University of Rochester Medical Center, 601
Elmwood Avenue, Box 672, Rochester, NY 14642, USA

Anónimo disse...

Em Portugal a única vacina obrigatória pelo estado português é a do Tétano, que é realmente uma doença perigosa, com morte dolorosa se não tratada, e que pode acontecer realmente no dia a dia, especialmente em zonas rurais, com má higiene.

Pessoalmente acho que dá-la a uma crinça já com 6 anos é uma hipótese mais aceitável do que dá-la a uma criança ,mais pequena.

Em Portugal há muita pressão social para vacinar. Toda a gente vacina porque não há informação e porque todas as escolas (menos as de Rudolf steiner) exigem a vacinação.

Se pensares bem, e em todas as doenças para as quais há vacinas no mercado apenas a varíola foi extinta... de cada vez que se vacinam milhares de crianças para o sarampo, surgem sempre casos graves de sarampo, e na população americana, há um risco de 10x mais alto de ter encefalite, que é a doença que pode eventualmente surgir com o sarampo.... pode se a tiveres de forma natural, não de forma forçada, com a vacina, entendes?
O que se passa em Portugal e é muito grave são as idades e os conservantes. Aqui, um bebé quando nasce leva logo três vacinas, entre elas, a tuberculose, que não dá imunidade desde há mais de 20 anos, e a hepatite B que apenas se apanha através de fluidos sexuais ou comportamentos de risco (drug addicts and prostituts), então para quê dar vacinas a bebés? Um bebé NÂO tem sistema imunitário e ao receber doenças directamente no sangue (que não entram normalmente pelo sangue - directo) tem uma reação forte e o seu sistema imunitário fica debilitado, fraco.

No primeiro ano de vida de um bebé ele recebe cerca de 22 doses de cerca 12 vacinas (se não me engano) para muitas doenças que talvez o bebé nunca apanhe e que talvez só precise de se proteger quando é adolescente...

Outro problema grave é que em portugal as vacinas são conservadas com produtos proibidos como o thimeresol, que servia para limpar petróleo ou gasolina... e hoje em dia nem os veterinários dão vacinas dessas aos animais nos EUA. São tb conservadas em alumínio que dá problemas a nível dos gânglios linfácticos e têm surgido cada vez mais doenças de sangue em crianças pequenas...
Para não falar em lactose, gema de ovo, etc etc.

Aquilo que mais me aborrece por cá é a pressão para vacinar bebés pequenos, muito pequenos para serem vacinados, e depois as condições para vacinar. Acredito que se o meu filho tiver uma boa higiene, uma boa alimentação, muito colo e amor, que pode ficar doente mas tb fica melhor mais depressa, entendes!? Eu não vacinei o Simão por várias razões, mas especialmente porque teve reações muito fortes depois de uma dose de vacina, mas ás vezes tenho medo e penso: e se ele apanhar uma doença porque não está vacinado?... mas se ele estiver vacinado, tb pode apanhar a doença, pela vacina ou porque ela não funcionou, e pode ainda por cima ficar com a sáude mais débil porque as levou...
Acho que vacinar dá uma falsa sensação de segurança...

Anónimo disse...

Maria
Precisa de ler este livro urgentemente. Hoje dormi apenas 4 horas à conta da leitura do livro "Memórias de um Homem de Vidro"...Comecei ontem e já vou a meio. Fiquei encantada, emocionada e definitivamente rendida a este testemundo de humanidade, ainda mais marcante por ter sido um médico a escrevê-lo.
Durante alguns anos convivi com médicos (na parte administrativa) e reconheci através deste relato a sua postura e muitas das relações que se estabelecem entre o pessoal hospitalar.
Ter ousado quebrar alguns dos dogmas sobre os quais se ergue a medicina contemporânea foi um acto de coragem e, acima de tudo, de muito amor e respeito pela Vida.

Unknown disse...

Olá querida amiga vim te visitar e trazer-te um abraço meu e um grande beijinho deixando para ti os votos de um feliz natal

Anónimo disse...

1. Vacinas são tóxicas
- Vacinas contêm substâncias que são tóxicas para o ser humano (mercúrio, formol, alumínio etc.) As bulas de vacinas contêm esta e outras informações que por lei devem estar disponíveis ao público. Apesar dessas bulas serem impressas para os consumidores, os médicos não as mostram a seus pacientes.
- Vacinas são cultivadas sobre tecidos estranhos e contêm material genético alterado de origem humana e animal.

2. A vacinação deprime e prejudica a função do cérebro e da imunidade.
Pesquisas científicas honestas e imparciais mostraram que a vacinação é fator de risco em muitas doenças, como:
- Síndrome de morte infantil súbita (SIDS)
- Disfunções de desenvolvimento (autismo, convulsões, retardo mental, hiperatividade, dislexia, etc.)
- Deficiência imunológica (AIDS, Síndrome Epstein Barre etc.).
- Doenças degenerativas (distrofia muscular, esclerose múltipla, artrite, câncer, leucemia, lúpus, fibromialgia etc.).

3. O alto índice de reações adversas a vacinas é ignorado e negado pela medicina convencional.
- Antes de 1990, os médicos não eram legalmente obrigados a notificar as reações adversas ao órgão de controle de doenças nos EUA ( CDC - US Centers for Disease Control).
- Reações adversas são consideradas “normais”, são ignoradas ou diagnosticadas como outras doenças. Apesar desse sistema precário, os danos notificados são numerosos.
- Apesar da obrigação legal atual, menos do que 10% dos médicos notificam ao CDC os danos que testemunham .
- Ao longo da história, muitos profissionais renomados da área da saúde, em todo o mundo, declararam sua oposição veemente à vacinação, chamando-a de fraude científico.

4. Programas de vacinação em massa expõem o público ao perigo de forma sistemática e irresponsável, desrespeitando os direitos da população.

5. Não há prova de que vacinas são seguras ou eficazes.
- Não há estudos com grupos de controle. Autoridades consideram que “não vacinar” é antiético e se recusam a estudar voluntários não vacinados. Se estudos de controle fossem realizados de acordo com procedimentos científicos honestos, a vacina seria proibida.
- Estudos realizados não estão eliminando o preconceito do leitor. As autoridades que reúnem e publicam estatísticas de doenças trabalham em conjunto com laboratórios que produzem as vacinas e têm interesses econômicos neles. Efeitos colaterais e óbitos são atribuídos a tudo menos vacinas para distorcer os resultados e fazer parecer que as vacinas têm algum mérito.

Anónimo disse...

6. As leis permitem que os laboratórios quebrem a confiança pública.
- Em processos particulares por danos causados pela vacina, a informação apresentada mostra que as vacinas podem ser letais.
- Fabricantes de vacinas impõem confidencialidade como instrumentos nos processos para impedir que o autor da ação divulgue a verdade sobre a perigosa natureza das vacinas. O governo permite o uso destas táticas antiéticas, que põe em risco a saúde pública.

7. Nos EUA, a lei de Lesões da Vacina Infantil de 1987 age como tranqüilizante
- Este programa de compensação finge reconhecer a existência de danos vacinais, “consertando” os erros cometidos. Nada nessa lei tenta impedir que tais ocorrências se repitam no futuro.
- Essa lei é o resultado da pressão dos fabricantes de vacinas para que sejam “imunizados” contra processos particulares que podem chegar a milhões de dólares por caso.
- Os fabricantes de vacinas conseguiram se eximir bem da responsabilidade e, nos anos recentes, a compensação ficou cada vez mais difícil através desse programa. Os parâmetros definindo o dano vacinal têm mudado e, em muitos casos, os pais são acusados pela Síndrome da Criança Sacudida.

8. Empresas de seguros, que fazem os melhores estudos de sinistros, abandonaram por completo as coberturas de danos à vida e à propriedade causados por:
- Ato de Deus
- Guerra nuclear e acidentes em usinas nucleares
- Vacinação.

9. Vacinação não é medicina de urgência.
- Afirmam que vacinas evitam um possível risco futuro. No entanto, as pessoas são pressionadas a decidirem na hora. O uso do medo e de intimidação pelo médico para forçar uma vacina é antiético. Vacinas são medicamentos com sérias reações adversas. Deveria haver tempo para reflexão antes de uma decisão.

10. Não há lei exigindo vacinações para bebês ou qualquer pessoa.
-A vacinação está ligada ao atendimento escolar, mas não é obrigatória. Isenções de vacinas, apesar de restritas e controladas, são inerentes a cada lei e podem ser expandidas por pressão pública.
- Nos EUA, os Ministérios da Saúde e da Educação e a Associação Médica Americana lucram com a venda de vacinas. Eles raramente divulgam a existência e detalhes das isenções.

(fonte: www.taps.org.br)

Existe MUITÍSSIMA literatura disponível, que comprova tudo o que foi dito aqui sobre as vacinas. Trabalhos científicos, trabalhos analisando os científicos e “traduzindo-os” para leigos compreeenderem; tem de tudo, e muito disponível na internet, principalmente em inglês. Em português tem pouquíssima coisa. Você pode buscar na internet pelas palavras “dangers of vaccines” e “mercury in vaccines”.

Mark Sircus (criador do IMVA), tem muita coisa escrita. O primeiro livro dele sobre o assunto , “Cry of the Heart” está agora disponível gratuitamente na net, pelo site dele www.worldpsychology.net (Se não conseguir o livro lá nos escreva e nós providenciaremos). Este livro, que ele escreveu para pais, não está num formato científico, com referências e tudo mais. Mas tem muita informação boa, vale a pena mesmo dar uma olhada. Inclusive lá tem as tabelas estatísticas que mostram como as doenças infantis NÃO foram erradicadas pelas vacinas. Além do livro ele tem vários artigos sobre o assunto, esses com referências científicas. Muitos foram enviados para esta lista VacinaVeritas, as mensagens estão todas arquivadas.

Anónimo disse...

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=11701571&dopt=Abstract


Deaths from chickenpox in England and Wales 1995-7: analysis of routine
mortality data.

Rawson H, Crampin A, Noah N.

Guy's King's College, St. Thomas's Hospital School of Medicine and
Dentistry, London, UK.

OBJECTIVE: To evaluate the epidemiology and impact of mortality from
chickenpox in England and Wales. DESIGN: Review of death certificates from
the Office for National Statistics on which codes for "chickenpox" or
"varicella" were mentioned. Further information ascertained from certifying
physician. PARTICIPANTS: Those certified as having died from chickenpox in
England and Wales, 1995-7. MAIN OUTCOME MEASURES: Diagnosis and age and sex
distributions of deaths from chickenpox. RESULTS: On average, 25 people a
year die from chickenpox. Overall case fatality was 9.22 per 100 000
consultations for chickenpox. Adults accounted for 81% of deaths and 19% of
consultations. Deaths were twice as common in men as in women. More of
those who died were born outside United Kingdom than expected (12% v 4%).
CONCLUSIONS: Chickenpox is not a mild disease. Deaths in adults are
increasing, both in number and proportion.

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Vaccine Dangers On-Line course - http://www.nccn.net/~wwithin/vaccineclass.htm
Homeopathy On-Line course - http://www.nccn.net/~wwithin/homeo.htm